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Then , we study the dynamics of a single electron spin in a DQD and its readout via QPC .

接着，我们重点研究了在耦合双量子点系统中单个电子自旋的动力学及如何通过运用一个量子点接触器件读出它的白旋状态。

In addition , the effects of the coupling strength between the DQD and the NAMR on these energy shifts are studied .

此外，量子点和纳米机械振子之间的耦合强度对这些能级移动的影响也被研究了。

DQD ( 20 - 200 μ mol / L ) inhibited the actin polymerization induced by thrombin ( 500 U / L ) in platelets in a concentration-dependent manner .

DQD（20-200μmol/L）对凝血酶诱导的血小板肌动蛋白聚合有较强的抑制作用。

DQD ( 10-160 μ mol / L ) inhibited the cytosolic Ca2 + / PL dependent PKC from platelets in a concentration-dependent manner , but had no effect on membrane PKC .

DQD（10-160μmol/L）抑制血小板胞浆Ca~（2+）/PL依赖的PKC，但DQD不影响胞膜PKC。

CONCLUSION : DQD inhibited pig platelet aggregation induced by thrombin and its molecular mechanism was due to its inhibition of Ca2 + influx , intracellular Ca2 + mobilization , Ca2 + /

结论：DQD对凝血酶诱导的猪血小板聚集有抑制作用，其分子作用机制是由于抑制血小板外钙内流、内钙动员、Ca~（2+）/PL依赖的PKC和肌动蛋白聚合。血小板；